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Messages - dirksilver

Pages: 1 ... 6 7 [8] 9 10 ... 13
106
Progress Journals & Experimental Routines / Re: ADARQ's journal
« on: April 06, 2011, 12:51:08 am »
i was digging those bounce off the backboard attempts man! i'd love to see more of that mon ami

also that facebook post was awesome! SCREW RAP MUSIC!!!

wtf

i'm not such a fan...particularly if i'm trying to get amped up

some rap is ok...but it gets so old so fast...even jay Z agrees

107
Progress Journals & Experimental Routines / Re: ADARQ's journal
« on: April 06, 2011, 12:42:14 am »
i was digging those bounce off the backboard attempts man! i'd love to see more of that mon ami

also that facebook post was awesome! SCREW RAP MUSIC!!!

108
Pics, Videos, & Links / Re: beast
« on: April 05, 2011, 10:40:13 pm »
maybe not beast but definitely pimp!


http://www.youtube.com/watch?v=1g8dmIhzyX8

109
SPANKED!

110
Pics, Videos, & Links / Re: Lithuanian dunk contest
« on: April 03, 2011, 05:35:52 pm »
Estonian dunk contest not Lithuanian  :uhhhfacepalm:

stupid raptor!

111
you also need some good form gripping the bar, you can't do that if squatting bugs your shoulders in the least.

peace

i guess i wont try this then hahaha

112
oh and yeah i dunno how much you should do it but i always thought walk outs with added weight was a good idea for potentiation

try it out man and elt us all know

113
Just do some occassional half or quarter squat reps or workouts and itll have the same effect. Supramaximal supports or walkouts can be very hard on the body if you're not real careful.  The only time I can remember acutely injuring myself in the weight room was doing this workout from Poliquin about 14 years ago:

http://vuesdumonde.forumactif.com/t7998-heavy-supports

He says to use 200% 1rm but I think I was only using 135%. I cant imagine anyone using a legit 200%.  On one set the bar shifted ever so slightly out of place and I had to struggle a bit to get it back on the rack. I never felt anything wrong during the workout but later that night and the next day I was literally glued to the bed and couldnt move....Id evidently severely strained a ligament in my back.

scarey chit kelly!

putting out a new article any time soon?

114
Pics, Videos, & Links / Re: Lithuanian dunk contest
« on: April 03, 2011, 02:06:25 pm »
nice! my family is from lithuania

115
Program Review / Re: Vertfreak
« on: April 02, 2011, 04:58:58 pm »
I never understood why you'd ever buy a program. There's the internet nowadays, tons of free info available, and when you think about it, it's not even that complicated. Get your squat up, practice your jumps and overload the movement with depth jumps/bounds/sprints and that's pretty much it. Get bodyfat down if it's high and that's it.

I didn't understand myself too, then. I admit, I was wrong. I wanted a shortcut. I wanted to get 4-8 inches within 3 months and I thought VF2.0 will give that. With the hype of Adam Link, having 50 inches vert,I thought this will give me my dream. I didn't know adarq.org by then. I only discovered this forum, 3 weeks after I bought the program. Still I was hoping to finish the program and gain the inches but abviuosly I didn't. I thought by then that what I was doing was great, and I was in the right path. But the results shows otherwise. I made my mistake and paying for it. :'(

I had VJB before VF2.0 but I was not contended because, frankly, I jsut downloaded VJB in torrent. And my gains are not consistent due to my poor consistency and have no faith in VJB thinking it was not enough and thinking there is a better alternative which is VF 2.0 that will give me better result. Then again I was wrong.

Owell, life is shit and hard. Need to learn from mistakes. Which is what I'm doing now. ;)
I jsut downloaded VJB in torrent

Just don't tell that to Kelly.

you guys and your lack of respect for intellectual property

theft is theft people...doesn't matter how you cut it

116
 

When It Comes to Building Tendons, Not All Steroids are Created Equal
   

by Hypertrophy
CEM-Meso.com

Stimulation of collagen synthesis by the anabolic steroid stanozolol.

Researchers: Falanga V, Greenberg AS, Zhou L, Ochoa SM, Roberts AB, Falabella A, Yamaguchi Y; University of Miami School of Medicine, Department of Dermatology, Miami, Veterans Affairs Medical Center, Florida, USA.

Source: J Invest Dermatol 1998 Dec;111(6):1193-7

Summary: In this report, we measured the effect of the anabolic steroid stanozolol on cell replication and collagen synthesis in cultures of adult human dermal fibroblasts. Stanozolol (0.625-5 micrograms per ml) had no effect on fibroblast replication and cell viability but enhanced collagen synthesis in a dose-dependent manner. Stanozolol also increased (by 2-fold) the mRNA levels of alpha1 (I) and alpha1 (III) procollagen and, to a similar extent, upregulated transforming growth factor-beta1 (TGF-beta1) mRNA and peptide levels. There was no stimulation of collagen synthesis by testosterone. The stimulatory effects of stanozolol on collagen synthesis were blocked by a TGF-beta1 anti-sense oligonucleotide, by antibodies to TGF-beta, and in dermal fibroblast cultures derived from TGF-beta-1 knockout mice. We conclude that collagen synthesis is increased by the anabolic steroid stanozolol and that, for the most part, this effect is due to TGF-beta-1. These findings point to a novel mechanism of action of anabolic steroids.

Discussion: I must first acknowledge that the commonly held belief is that anabolic steroids predispose an athlete to tendon rupture. This conclusion is drawn from animal studies showing that some steroids produce a larger, stiffer tendon in rats and that these steroid-induced tendons "fail" before the tendons from the control animals. The term fail refers to the breaking point.

The interesting thing about the present study is that the steroid stanozolol (Winstrol) had a different effect than testosterone. If you are a regular reader of MESO-Rx you should be well aware that not all steroids act in the same manner. And that because of subtle differences in there molecular structure they are able to elicit different responses. For example, Deca seems to act primarily through the androgen receptor (AR) where as Dianabol has effects beyond those associated with the AR.

Because synthetic steroids have differ in their chemical properties it should not be surprising that testosterone did not have the same effect as Winstrol. Winstrol increased collagen synthesis as opposed to testosterone which did not in this study. Interpreting the results of this study are more difficult than simply describing them. Other researchers have suggested that steroids cause a rapid increase in protein synthesis within tendon fibroblasts which results in fibroids or fibrous nodules within the tendon (Michna,1988). These fibroids alter the mechanical properties of the tendon perhaps predisposing it to rupture. It is also noted that during short term use of steroids there is an alteration in the alignment of collagen fibers which may also lead to rupture. Interestingly these alterations in collagen metabolism are transient with markers of collagen turnover returning more or less to baseline after 3-4 weeks of steroid administration (Karpakka,1992). These same researchers noted that low dose anabolics effect primarily muscle collagenous tissue with tendon being effected only at higher doses (i.e. 5 times the therapeutic dose) which would more closely represent what is needed by bodybuilders to put on mass.

The question remains, dose this mean that Winstrol will actually help prevent tendon injury or will it lead to bigger yet stiffer tendons prone to injury? It is difficult to take animal research and extrapolate the results to humans. Stanozolol is used therapeutically in humans to treat a variety of connective tissue and vascular disorders and its clinical effects suggest that it can modulate connective tissue breakdown in people. Despite being labeled as "ineffective" by many bodybuilders it is very popular among athletes. As with most hormones, dosage plays a role in what effects are seen, be they positive or negative. Hopefully future studies will shed light on the therapeutic effects of different steroids on tendons in humans.

References:

Michna H Appearance and ultrastructure of intranuclear crystalloids in tendon fibroblasts induced by an anabolic steroid hormone in the mouse. Acta Anat (Basel) 1988;133(3):247-50

Karpakka JA, Pesola MK, Takala TE. The effects of anabolic steroids on collagen synthesis in rat skeletal muscle and tendon. A preliminary re

Read more from this MESO-Rx article at: http://www.mesomorphosis.com/articles/haycock/anabolic-steroids-and-collagen-synthesis.htm#ixzz1IDM8nfTZ


so basically this study says it doesn't know







basically, that study shows that winstrol is used to IMPROVE connective tissue in certain cases and you cant draw human conclusions based on rat research.  So basically, its just showing how the idiots who go around saying "winstrol causes tendon damage" got their flawed info from, basically.

i guess it's all acedemic anyways since i'm never going to use the stuff

117
anyone got an online link i can watch it from? i'm at work

Might be available on ESPN3.com since ESPN2 is airing it.

nothing on espn3.com

118
anyone got an online link i can watch it from? i'm at work

119
i think he could...some of those dunks on that vid are insane!

120
 

When It Comes to Building Tendons, Not All Steroids are Created Equal
   

by Hypertrophy
CEM-Meso.com

Stimulation of collagen synthesis by the anabolic steroid stanozolol.

Researchers: Falanga V, Greenberg AS, Zhou L, Ochoa SM, Roberts AB, Falabella A, Yamaguchi Y; University of Miami School of Medicine, Department of Dermatology, Miami, Veterans Affairs Medical Center, Florida, USA.

Source: J Invest Dermatol 1998 Dec;111(6):1193-7

Summary: In this report, we measured the effect of the anabolic steroid stanozolol on cell replication and collagen synthesis in cultures of adult human dermal fibroblasts. Stanozolol (0.625-5 micrograms per ml) had no effect on fibroblast replication and cell viability but enhanced collagen synthesis in a dose-dependent manner. Stanozolol also increased (by 2-fold) the mRNA levels of alpha1 (I) and alpha1 (III) procollagen and, to a similar extent, upregulated transforming growth factor-beta1 (TGF-beta1) mRNA and peptide levels. There was no stimulation of collagen synthesis by testosterone. The stimulatory effects of stanozolol on collagen synthesis were blocked by a TGF-beta1 anti-sense oligonucleotide, by antibodies to TGF-beta, and in dermal fibroblast cultures derived from TGF-beta-1 knockout mice. We conclude that collagen synthesis is increased by the anabolic steroid stanozolol and that, for the most part, this effect is due to TGF-beta-1. These findings point to a novel mechanism of action of anabolic steroids.

Discussion: I must first acknowledge that the commonly held belief is that anabolic steroids predispose an athlete to tendon rupture. This conclusion is drawn from animal studies showing that some steroids produce a larger, stiffer tendon in rats and that these steroid-induced tendons "fail" before the tendons from the control animals. The term fail refers to the breaking point.

The interesting thing about the present study is that the steroid stanozolol (Winstrol) had a different effect than testosterone. If you are a regular reader of MESO-Rx you should be well aware that not all steroids act in the same manner. And that because of subtle differences in there molecular structure they are able to elicit different responses. For example, Deca seems to act primarily through the androgen receptor (AR) where as Dianabol has effects beyond those associated with the AR.

Because synthetic steroids have differ in their chemical properties it should not be surprising that testosterone did not have the same effect as Winstrol. Winstrol increased collagen synthesis as opposed to testosterone which did not in this study. Interpreting the results of this study are more difficult than simply describing them. Other researchers have suggested that steroids cause a rapid increase in protein synthesis within tendon fibroblasts which results in fibroids or fibrous nodules within the tendon (Michna,1988). These fibroids alter the mechanical properties of the tendon perhaps predisposing it to rupture. It is also noted that during short term use of steroids there is an alteration in the alignment of collagen fibers which may also lead to rupture. Interestingly these alterations in collagen metabolism are transient with markers of collagen turnover returning more or less to baseline after 3-4 weeks of steroid administration (Karpakka,1992). These same researchers noted that low dose anabolics effect primarily muscle collagenous tissue with tendon being effected only at higher doses (i.e. 5 times the therapeutic dose) which would more closely represent what is needed by bodybuilders to put on mass.

The question remains, dose this mean that Winstrol will actually help prevent tendon injury or will it lead to bigger yet stiffer tendons prone to injury? It is difficult to take animal research and extrapolate the results to humans. Stanozolol is used therapeutically in humans to treat a variety of connective tissue and vascular disorders and its clinical effects suggest that it can modulate connective tissue breakdown in people. Despite being labeled as "ineffective" by many bodybuilders it is very popular among athletes. As with most hormones, dosage plays a role in what effects are seen, be they positive or negative. Hopefully future studies will shed light on the therapeutic effects of different steroids on tendons in humans.

References:

Michna H Appearance and ultrastructure of intranuclear crystalloids in tendon fibroblasts induced by an anabolic steroid hormone in the mouse. Acta Anat (Basel) 1988;133(3):247-50

Karpakka JA, Pesola MK, Takala TE. The effects of anabolic steroids on collagen synthesis in rat skeletal muscle and tendon. A preliminary re

Read more from this MESO-Rx article at: http://www.mesomorphosis.com/articles/haycock/anabolic-steroids-and-collagen-synthesis.htm#ixzz1IDM8nfTZ


so basically this study says it doesn't know





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